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Nanomaterials can induce genomic alterations and facilitate intracellular generation of hydroxyl radicals, resulting in increased reactive oxygen species and DNA damage

We propose that curcumin exerts its broad therapeutic effects via a unified inflammationoxidative stress axis, primarily by inhibiting NF-B and NLRP3 activation, reducing ROS and pro-inflammatory cytokines (TNF-, IL-1/18), as seen across models of depression, metabolic disorders, and neurodegeneration